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Annotated account of digestion - 16 references
Outline the mechanisms by which ATP is synthesised in chloroplasts and in mitochondria. What are the fundamental similarities and what are the most important differences between ATP synthesis in chloroplasts and mitochondria. - 4 references
Are DMDs the most effective treatment for treating Multiple Sclerosis - 8 references
The Impacts of Wildfires on Caribou in the NWT - 10 references
Sonic Hedgehog Signalling - 9 references
Shai’Ann Simpson Assignment 2: Questions on Phenylalanine in newborns 1. The condition the baby has in high levels of phenylalanine in the blood. High levels of this amino acid cause a defect in the body. Amino acids help build protein, and the buildup of this amino acid is what causes the defects. 2. PKU is inherited so it must be passed down by both parents. Babies with this disease lack the phenylalanine hydroxylase enzyme which breaks down the phenylalanine in the body. Without this break down of phenylalanine, it can cause harmful damage to the central nervous system and/or cause brain damage. This amino acid is found in foods and newborns with this buildup have strict diets to control their intake of phenylalanine. 3. Classical PKU is inherited in an autosomal recessive manner and is the result of mutations in the PAH gene. Most variation in classical PKU is due to heterogeneity in the mutant alleles. 4. This disease does not effect a specific sex, it is mainly dependent on whether or not both of its parents pass down the gene change to have PKU, since it is inherited. If only one parent has PKU, then the child only is a carrier of PKU. 5. This disease is treated by having a controlled diet with foods that are low in phenylalanine. Control of the diet is supposed to be followed throughout the person’s whole life even through pregnancy when or if the child is females and becomes pregnant. Newborns are to be tested of their phenylalanine levels weekly and the number of visits decrease to biweekly and then monthly. Symptoms in newborns and as they age tend to show intellectual disorders, delayed social skills, skin/hair/eyes discoloration, disproportions in the body parts, taking longer to eat, sit, talk etc. 6. Gaucher’s disease is an inherited disorder caused by a missing enzyme called glucocerebrosidase. The buildup of too much fatty substances can cause organ failures in your body. Lack of this enzyme can cause abnormal enlargement of the spleen and/or liver in type 1 of this disease, luckily this type does not affect the brain. In type 2, severe brain damage can arose and also appear in infants. Type 3 of this disease is like type 1, where you will see augmentation of the liver and spleen and maybe brain damage as well. This all usually starts in early childhood. There is no cure for this disease, only treatments of medicine and enzyme replacement therapy for type 1 and 3, with rather scarce options of treatment for type 2 and type 3 brain damage. - 4 references
Bacterial sources of biofuels and the history of biofuels - 10 references
Dendrobates tinctorius - 3 references
Treatments for decompressed smallmouth bass - 13 references
digoxin - 3 references
calcium oscillations on hepatocyte cells fluo 4 AM - 3 references
Variety of Anura, Caudata and Caecilians Parental Care - 5 references
Sonic hedgehog - 8 references
Developmet of HIV database - 10 references
monkeypox - 9 references
Sumatran Tiger Conservation - 26 references
difference between liver cells and nerve cells - 3 references
Ihr Literaturverzeichnis - 23 references
inherited and infectious disease - 14 references
Tamiflu as a treatment for influenza - 5 references
P and P - 24 references
West nile virus - 11 references
reapiration - 8 references
Ethical issues arising from the termination of Down syndrome foetuses - 17 references
Maggot - 8 references

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